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As a mitochondrial-derived peptide, it activates AMPK pathways associated with improved glucose metabolism and metabolic flexibility

doi: 10.18632/aging.203506 68 JinYQiuJLuXMaYLiG

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SBI are characterized by a range of pathological alteration, including mitochondrial dysfunction and oxidative stress, which are critical contributors to neuronal death and ongoing disease progression [7,8,9]

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