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A growing number of studies examining how dysbiosis might drive NAFLD have identified a number of plausible mechanisms, including changes in short-chain fatty acids (SCFAs) metabolism, increased intestinal permeability and lipopolysaccharides (LPS) activation of toll-like receptors (TLRs) and inflammasomes, endogenous ethanol production, decreased choline availability, and trimethylamine production (69)

we can now say that these chemicals are found in the body of human adolescents who use these products, they wrote in their report, published in the journal Pediatrics

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About the author Kate Thompson is a lifestyle writer who has been widely published for over 20 years in digital content